Pressure Ulcers (Pressure Sores) | dermaproblems.com
Pressure ulcers develop at body-support interfaces over bony prominences as a result of external compression of the skin, shear forces, and friction, which produce ischemic tissue necrosis. Pressure ulcers occur in patients who are obtunded mentally or have diminished sensation (as in spinal cord disease) in the affected region. Secondary infection results in localized cellulitis, which can extend locally into bone or muscle or into the bloodstream with resultant bacteremia and sepsis.
External compression of the dermis and hypodermis leads to ischemic tissue damage and necrosis causing by Pressure Ulcers. The mainly and most risky factors for developing pressure ulcers are as they follow: inadequate nursing care, diminished sensation/immobility (obtunded mental status, spinal cord disease), hypotension, fecal or urinary incontinence, presence of fracture, hypoalbuminemia, and poor nutritional status. The mean skin capillary pressure is approximately 25 mmHg. External compression with pressures <30 mmHg occludes the blood vessels so that the surrounding tissues become anoxic. Amount of damage is proportional to extent and duration of pressure.
Sepsis Pressure Ulcers - Bookshelf
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